| [3] |
Chistiakov DA, Myasoedova VA, Revin VV, et al. The impact of interferon-regulatory factors to macrophage differentiation and polarization into M1 and M2[J]. Immunobiology, 2018, 223(1): 101-111.
|
| [4] |
Chavez-Galan L, Olleros ML, Vesin D, et al. Much more than M1 and M2 macrophages, there are also CD169(+) and TCR(+) macrophages[J]. Front Immunol, 2015, 6: 263.
|
| [5] |
Burgess M, Wicks K, Gardasevic M, et al. Cx3CR1 expression identifies distinct macrophage populations that contribute differentially to inflammation and repair[J]. Immunohorizons, 2019, 3(7): 262-273.
|
| [6] |
Minutti CM, Knipper JA, Allen JE, et al. Tissue-specific contribution of macrophages to wound healing[J]. Semin Cell Dev Biol, 2017, 61: 3-11.
|
| [7] |
Kimball A, Schaller M, Joshi A, et al. Ly6C(Hi) blood monocyte/macrophage drive chronic inflammation and impair wound healing in diabetes mellitus[J]. Arterioscler Thromb Vasc Biol, 2018, 38(5): 1102-1114.
|
| [8] |
Rodero MP, Hodgson SS, Hollier B, et al. Reduced Il17a expression distinguishes a Ly6c(lo)MHCII(hi) macrophage population promoting wound healing[J]. J Invest Dermatol, 2013, 133(3): 783-792.
|
| [9] |
Chen H, Shi R, Luo B, et al. Macrophage peroxisome proliferator-activated receptor γ deficiency delays skin wound healing through impairing apoptotic cell clearance in mice[J]. Cell Death Dis, 2015, 6(1): e1597.
|
| [10] |
Goren I, Müller E, Schiefelbein D, et al. Systemic anti-TNFalpha treatment restores diabetes-impaired skin repair in ob/ob mice by inactivation of macrophages[J]. J Invest Dermatol, 2007, 127(9): 2259-2267.
|
| [11] |
Wang Q, Zhu G, Cao X, et al. Blocking AGE-RAGE signaling improved functional disorders of macrophages in diabetic wound[J]. J Diabetes Res, 2017, 2017: 1428537.
|
| [12] |
Schultze JL, Schmieder A, Goerdt S. Macrophage activation in human diseases[J]. Semin Immunol, 2015, 27(4): 249-256.
|
| [13] |
Talamonti E, Pauter AM, Asadi A, et al. Impairment of systemic DHA synthesis affects macrophage plasticity and polarization: implications for DHA supplementation during inflammation[J]. Cell Mol Life Sci, 2017, 74(15): 2815-2826.
|
| [14] |
Jia YC, Qiu S, Xu J, et al. Docosahexaenoic acid improves diabetic wound healing in a rat model by restoring impaired plasticity of macrophage progenitor cells[J]. Plast Reconstr Surg, 2020, 145(5): 942e-950e.
|
| [15] |
Wilkinson HN, Clowes C, Banyard KL, et al. Elevated local senescence in diabetic wound healing is linked to pathological repair via CXCR2[J]. J Invest Dermatol, 2019, 139(5): 1171-1181.
|
| [16] |
山慧，张子锐，王晓莹，等. 去铁胺对深部组织损伤小鼠巨噬细胞极化和创面愈合的调节机制[J]. 中华烧伤与创面修复杂志，2022, 38(8): 767-777.
|
| [17] |
王璐，顾建英，卫传元，等. 间充质干细胞诱导巨噬细胞极化促进创面愈合的研究进展[J]. 中华整形外科杂志，2019, 35(12): 1275-1278.
|
| [18] |
石志远，张博涵，孙佳辰，等. 表皮干细胞在皮肤创面修复中的作用与机制研究进展[J]. 中华烧伤与创面修复杂志，2022, 38(9): 854-858.
|
| [1] |
Powers JG, Higham C, Broussard K, et al. Wound healing and treating wounds: chronic wound care and management[J]. J Am Acad Dermatol, 2016, 74(4): 607-25.
|
| [2] |
Xu X, Gu S, Huang X, et al. The role of macrophages in the formation of hypertrophic scars and keloids[J]. Burns Trauma, 2020, 8: tkaa006.
|
| [19] |
Ahangar P, Mills SJ, Cowin AJ. Mesenchymal stem cell secretome as an emerging cell-free alternative for improving wound repair[J]. Int J Mol Sci, 2020, 21(19): 7038.
|
| [20] |
Uchiyama A, Motegi SI, Sekiguchi A, et al. Mesenchymal stem cells-derived MFG-E8 accelerates diabetic cutaneous wound healing[J]. J Dermatol Sci, 2017, 86(3): 187-197.
|
| [21] |
Németh K, Leelahavanichkul A, Yuen PS, et al. Bone marrow stromal cells attenuate sepsis via prostaglandin E(2)-dependent reprogramming of host macrophages to increase their interleukin-10 production[J]. Nat Med, 2009, 15(1): 42-49.
|
| [22] |
Liu W, Yu M, Xie D, et al. Melatonin-stimulated MSC-derived exosomes improve diabetic wound healing through regulating macrophage M1 and M2 polarization by targeting the PTEN/AKT pathway[J]. Stem Cell Res Ther, 2020, 11(1): 259.
|
| [23] |
Wang P, Theocharidis G, Vlachos IS, et al. Exosomes derived from epidermal stem cells improve diabetic wound healing[J]. J Invest Dermatol, 2022, 142(9): 2508-2517. e13.
|
| [24] |
Li J, Wei C, Yang Y, et al. Apoptotic bodies extracted from adipose mesenchymal stem cells carry microRNA-21-5p to induce M2 polarization of macrophages and augment skin wound healing by targeting KLF6[J]. Burns, 2022, 48(8): 1893-1908.
|
| [25] |
He X, Dong Z, Cao Y, et al. MSC-derived exosome promotes M2 polarization and enhances cutaneous wound healing[J]. Stem Cells Int, 2019, 2019: 7132708.
|
| [26] |
Lee JM, Govindarajah V, Goddard B, et al. Obesity alters the long-term fitness of the hematopoietic stem cell compartment through modulation of Gfi1 expression[J]. J Exp Med, 2018, 215(2): 627-644.
|
| [27] |
Chen S, Li R, Cheng C, et al. Pseudomonas aeruginosa infection alters the macrophage phenotype switching process during wound healing in diabetic mice[J]. Cell Biol Int, 2018, 42(7): 877-889.
|
| [28] |
Ramakant P, Verma AK, Misra R, et al. Changing microbiological profile of pathogenic bacteria in diabetic foot infections: time for a rethink on which empirical therapy to choose？[J]. Diabetologia, 2011, 54(1): 58-64.
|
| [29] |
Cavaillon JM. Exotoxins and endotoxins: Inducers of inflammatory cytokines[J]. Toxicon, 2018, 149: 45-53.
|
| [30] |
Goldufsky J, Wood SJ, Jayaraman V, et al. Pseudomonas aeruginosa uses T3SS to inhibit diabetic wound healing[J]. Wound Repair Regen, 2015, 23(4): 557-564.
|
| [31] |
Mariottoni P, Jiang SW, Prestwood CA, et al. Single-cell RNA sequencing reveals cellular and transcriptional changes associated with M1 macrophage polarization in hidradenitis suppurativa[J]. Front Med (Lausanne), 2021, 8: 665873.
|
| [32] |
Castro-Dopico T, Fleming A, Dennison TW, et al. GM-CSF calibrates macrophage defense and wound healing programs during intestinal infection and inflammation[J]. Cell Rep, 2020, 32(1): 107857.
|
| [33] |
Zhao H, Huang J, Li Y, et al. ROS-scavenging hydrogel to promote healing of bacteria infected diabetic wounds[J]. Biomaterials, 2020, 258: 120286.
|
| [34] |
Xu Z, Deng B, Wang X, et al. Nanofiber-mediated sequential photothermal antibacteria and macrophage polarization for healing MRSA-infected diabetic wounds[J]. J Nanobiotechnology, 2021, 19(1): 404.
|
| [35] |
Wu R X, Ma C, Liang Y, et al. ECM-mimicking nanofibrous matrix coaxes macrophages toward an anti-inflammatory phenotype: cellular behaviors and transcriptome analysis[J]. Appl Mater Today, 2020, 18: 100508.
|
| [36] |
Brouki Milan P, Pazouki A, Joghataei MT, et al. Decellularization and preservation of human skin: a platform for tissue engineering and reconstructive surgery[J]. Methods, 2020, 171: 62-67.
|
| [37] |
He C, Yang Z, Jin Y, et al. ADM scaffolds generate a pro-regenerative microenvironment during full-thickness cutaneous wound healing through M2 macrophage polarization via lamtor1[J]. Front Physiol, 2018, 9: 657.
|
| [38] |
Wu P, Liang Y, Sun G, et al. Engineering immune-responsive biomaterials for skin regeneration[J]. Biomater Transl, 2021, 2(1): 61-71.
|
| [39] |
Lazarus BS, Chadha C, Velasco-Hogan A, et al. Engineering with keratin: a functional material and a source of bioinspiration[J]. iScience, 2021, 24(8): 102798.
|
| [40] |
Waters M, Vandevord P, Van Dyke M. Keratin biomaterials augment anti-inflammatory macrophage phenotype in vitro[J]. Acta Biomater, 2018, 66: 213-223.
|
| [41] |
Qian Y, Zheng Y, Jin J, et al. Immunoregulation in diabetic wound repair with a photoenhanced glycyrrhizic acid hydrogel scaffold[J]. Adv Mater, 2022, 34(29): e2200521.
|
| [42] |
朱萌，陈禹州，区锦钊，等. 水溶性壳聚糖水凝胶对糖尿病小鼠感染全层皮肤缺损创面的作用及其机制[J]. 中华烧伤与创面修复杂志，2022, 38(10): 923-931.
|
| [43] |
卢毅飞，邓君，王竞，等. 乳酸乳球菌温敏水凝胶对糖尿病小鼠全层皮肤缺损创面愈合的影响及其机制[J]. 中华烧伤杂志，2020, 36(12): 1117-1129.
|
| [44] |
Zhao X, Pei D, Yang Y, et al. Green tea derivative driven smart hydrogels with desired functions for chronic diabetic wound treatment[J]. Adv Funct Mater, 2021, 31(18): 2009442.
|
| [45] |
Guo Y, Lin C, Xu P, et al. AGEs induced autophagy impairs cutaneous wound healing via stimulating macrophage polarization to M1 in diabetes[J]. Sci Rep, 2016, 6: 36416.
|
| [46] |
陈祺. 自噬活性对巨噬细胞极化与糖尿病小鼠创面愈合的影响[D]. 广州中医药大学，2019.
|
| [47] |
葛嘉媛. 巨噬细胞极化与自噬关系在糖尿病创面愈合中的影响研究[D]. 广州中医药大学，2020.
|
| [48] |
Feng X, Ji Y, Zhang C, et al. CCL6 promotes M2 polarization and inhibits macrophage autophagy by activating PI3-kinase/Akt signalling pathway during skin wound healing[J]. Exp Dermatol, 2023, 32(4): 403-412.
|
| [49] |
Ren H, Zhao F, Zhang Q, et al. Autophagy and skin wound healing[J]. Burns Trauma, 2022, 10: tkac003.
|
| [50] |
Kawano A, Ariyoshi W, Yoshioka Y, et al. Docosahexaenoic acid enhances M2 macrophage polarization via the p38 signaling pathway and autophagy[J]. J Cell Biochem, 2019, 120(8): 12604-12617.
|
| [51] |
Ji X, Jin P, Yu P, et al. Autophagy ameliorates pseudomonas aeruginosa-infected diabetic wounds by regulating the toll-like receptor 4/myeloid differentiation factor 88 pathway[J]. Wound Repair Regen, 2023, 31(3): 305-320.
|
| [52] |
Saitoh T, Akira S. Regulation of inflammasomes by autophagy[J]. J Allergy Clin Immunol, 2016, 138(1): 28-36.
|
| [53] |
Cao L, Wang Y, Wang Y, et al. Resolvin D2 suppresses NLRP3 inflammasome by promoting autophagy in macrophages[J]. Exp Ther Med, 2021, 22(5): 1222.
|
| [54] |
Cao Y, Chen J, Ren G, et al. Punicalagin prevents inflammation in LPS-induced RAW264. 7 macrophages by inhibiting FoxO3a/autophagy signaling pathway[J]. Nutrients, 2019, 11(11): 2794.
|
| [55] |
Takahama M, Akira S, Saitoh T. Autophagy limits activation of the inflammasomes[J]. Immunol Rev, 2018, 281(1): 62-73.
|
| [56] |
Maguire EM, Pearce SWA, Xiao Q. Foam cell formation: a new target for fighting atherosclerosis and cardiovascular disease[J]. Vascul Pharmacol, 2019, 112: 54-71.
|
| [57] |
王鹏，尹斌，苏映军，等. 结核分枝杆菌介导泡沫细胞形成机制的研究进展[J]. 中华微生物学和免疫学杂志，2020, 40(2): 154-159.
|
| [58] |
Orekhov AN, Nikiforov NG, Sukhorukov VN, et al. Role of phagocytosis in the pro-inflammatory response in LDL-induced foam cell formation; a transcriptome analysis[J]. Int J Mol Sci, 2020, 21(3): 817.
|
| [59] |
Moore KJ, Sheedy FJ, Fisher EA. Macrophages in atherosclerosis: a dynamic balance[J]. Nat Rev Immunol, 2013, 13(10): 709-21.
|
| [60] |
Wang X, Cao K, Sun X, et al. Macrophages in spinal cord injury: phenotypic and functional change from exposure to myelin debris[J]. Glia, 2015, 63(4): 635-651.
|
| [61] |
王鹏，尹斌，贾赤宇，等. 免疫荧光双重染色对临床结核性创面石蜡组织中泡沫细胞及结核分枝杆菌显示效果的比较分析[J]. 中华烧伤杂志，2021, 37(2): 157-163.
|
| [62] |
Tamoutounour S, Guilliams M, Montanana Sanchis F, et al. Origins and functional specialization of macrophages and of conventional and monocyte-derived dendritic cells in mouse skin[J]. Immunity, 2013, 39(5): 925-938.
|
| [63] |
Ahluwalia PK, Pandey RK, Sehajpal PK, et al. Perturbed microRNA expression by mycobacterium tuberculosis promotes macrophage polarization leading to pro-survival foam cell[J]. Front Immunol, 2017, 8: 107.
|
| [64] |
Bouhlel MA, Derudas B, Rigamonti E, et al. PPARgamma activation primes human monocytes into alternative M2 macrophages with anti-inflammatory properties[J]. Cell Metab, 2007, 6(2): 137-143.
|
| [65] |
Konger RL, Derr-Yellin E, Zimmers TA, et al. Epidermal PPARγ is a key homeostatic regulator of cutaneous inflammation and barrier function in mouse skin[J]. Int J Mol Sci, 2021, 22(16): 8634.
|
| [66] |
Luo Y, Tanigawa K, Kawashima A, et al. The function of peroxisome proliferator-activated receptors PPAR-γ and PPAR-δ in Mycobacterium leprae-induced foam cell formation in host macrophages[J]. PLoS Negl Trop Dis, 2020, 14(10): e0008850.
|
| [67] |
Riahi Y, Kaiser N, Cohen G, et al. Foam cell-derived 4-hydroxynonenal induces endothelial cell senescence in a TXNIP-dependent manner[J]. J Cell Mol Med, 2015, 19(8): 1887-1899.
|
| [68] |
Chen X, Deng Z, Feng J, et al. Necroptosis in macrophage foam cells promotes fat graft fibrosis in mice[J]. Front Cell Dev Biol, 2021, 9: 651360.
|
| [69] |
Johansen MD, Hortle E, Kasparian JA, et al. Analysis of mycobacterial infection-induced changes to host lipid metabolism in a zebrafish infection model reveals a conserved role for LDLR in infection susceptibility[J]. Fish Shellfish Immunol, 2018, 83: 238-242.
|
| [70] |
Wang P, Yin B, Zhang Z, et al. Foamy macrophages potentially inhibit tuberculous wound healing by inhibiting the TLRs/NF-κB signalling pathway[J]. Wound Repair Regen, 2022, 30(3): 376-396.
|